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Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation

Increasing evidence suggests that these cytokines influence bone metabolism through multiple mechanisms, promoting bone resorption while inhibiting bone formation, thereby serving as key drivers of osteoporosis onset and progression ( Figure 3 )

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Tryptophan can also be made into niacin via a de novo synthesis pathway, which adds to the functional redundancy